REVISED 1 2 The TRPV 4 channel is a novel regulator of intracellular Ca 2 + 3 in human esophageal epithelial cells 4 5

نویسندگان

  • Takashi Ueda
  • Michiko Shikano
  • Takeshi Kamiya
  • Takashi Joh
  • Shinya Ugawa
چکیده

REVISED 1 2 The TRPV4 channel is a novel regulator of intracellular Ca 3 in human esophageal epithelial cells 4 5 Takashi Ueda, Michiko Shikano, Takeshi Kamiya, Takashi Joh, and Shinya Ugawa 6 7 Department of Neurobiology and Anatomy, and Gastroenterology and Metabolism, 8 Graduate School of Medical Sciences, Nagoya City University, Nagoya, Japan 9 10 Running head: TRPV4 in human esophageal epithelial cells 11 12 Corresponding Author: 13 Takashi Ueda, Ph.D., 14 Department of Neurobiology and Anatomy, 15 Graduate School of Medical Sciences, 16 Nagoya City University, 17 1 Kawasumi, Mizuho-cho, Mizuho-ku, 18 Nagoya, Aichi 467-8601, Japan. 19 Tel: +81-52-853-8126 20 Fax: +81-52-852-8887 21 E-mail: [email protected] 22 23 Articles in PresS. Am J Physiol Gastrointest Liver Physiol (April 14, 2011). doi:10.1152/ajpgi.00511.2010

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

منابع مشابه

The TRPV4 channel is a novel regulator of intracellular Ca2+ in human esophageal epithelial cells.

The esophageal epithelium has sensory properties that enable it to sustain normal barrier function. Transient receptor potential vanilloid 4 (TRPV4) is a Ca(2+)-permeable channel that is activated by extracellular hypotonicity, polyunsaturated fatty acids, phorbol esters, and elevated temperature. We found that TRPV4 is expressed in both human esophageal tissue and in HET-1A cells, a human esop...

متن کامل

Protease-activated receptor-1 stimulates Ca(2+)-dependent Cl(-) secretion in human intestinal epithelial cells.

The thrombin receptor, protease-activated receptor-1 (PAR-1), has wide tissue distribution and is involved in many physiological functions. Because thrombin is in the intestinal lumen and mucosa during inflammation, we sought to determine PAR-1 expression and function in human intestinal epithelial cells. RT-PCR showed PAR-1 mRNA expression in SCBN cells, a nontransformed duodenal epithelial ce...

متن کامل

RNA interference targeted to multiple P2X receptor subtypes attenuates zinc-induced calcium entry.

A postulated therapeutic avenue in cystic fibrosis (CF) is activation of Ca(2+)-dependent Cl(-) channels via stimulation of Ca(2+) entry from extracellular solutions independent of CFTR functional status. We have shown that extracellular zinc and ATP induce a sustained increase in cytosolic Ca(2+) in human airway epithelial cells that translates into stimulation of sustained secretory Cl(-) tra...

متن کامل

The Level of Mesenchymal-Epithelial Transition Autophosphorylation is Correlated with Esophageal Squamous Cell Carcinoma Migration

Background: The MET receptor is a critical member of cancer-associated RTKs and plays an important role in different biological activities, including differentiation, migration, and cell proliferation. Methods: In this study, novel MET inhibitors were introduced and applied on esophageal squamous carcinoma cell line KYSE-30, and the level of proliferation and migration, as well as the activated...

متن کامل

An environmental sensor, TRPV4 is a novel regulator of intracellular Ca2+ in human synoviocytes.

The activation of a vanilloid type 4 transient receptor potential channel (TRPV4) has an obligatory role in regulation of intracellular Ca(2+) (Ca(2+)(i)) in several types of cells including vascular and sensory organs. In this study, we provide evidence that TRPV4 is a functional regulator of Ca(2+)(i) in human synoviocytes. Although significant expression of TRPV4 in synoviocytes from patient...

متن کامل

ذخیره در منابع من


  با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

عنوان ژورنال:

دوره   شماره 

صفحات  -

تاریخ انتشار 2011